Publication: 補充支鏈胺基酸與精胺酸對高強度間歇運動後恢復與後續運動表現的影響
| dc.contributor.advisor | 張振崗 | |
| dc.contributor.advisor | Chang, Chen-Kang | |
| dc.creator | 張家銘 | |
| dc.creator | Chang, Chai-Ming | |
| dc.date | 2010 | |
| dc.date.accessioned | 2017-02-27T06:31:17Z | |
| dc.date.accessioned | 2025-07-30T15:30:03Z | |
| dc.date.available | 2017-02-27T06:31:17Z | |
| dc.date.issued | 2017-02-27T06:31:17Z | |
| dc.description | 學位類別:碩士 | |
| dc.description | 校院名稱:國立台灣體育大學 | |
| dc.description | 系所名稱:競技運動學系碩士班 | |
| dc.description | 學號:19604020 | |
| dc.description | 畢業學年度:98年 | |
| dc.description | 論文頁數:66頁 | |
| dc.description.abstract | 支鏈胺基酸和精胺酸對於人體有許多生理功能,補充支鏈胺基酸可能可以增加胰島素反應,進而促進肌肉肝醣回補,可能可以降低骨骼肌中的蛋白質分解;補充精胺酸可能可以促進血管擴張降低運動時血液中所堆積的氨和乳酸,進而延緩肌肉疲勞,增進運動表現。本研究目的為探討補充支鏈胺基酸與精胺酸,對高強度間歇運動後之恢復期與及後續運動表現的影響,並探討其可能的生化機轉。本研究以9名男性大學角力選手為受試者,每名受試者皆以隨機順序進行3個trial,每個trial包含3次運動,每次運動包含3階段,受試者每階段於腳踏車測功計重覆10秒全力衝刺及20秒休息,在全力衝刺期間阻力設為0.1 kp/kg,每階段包含4次的間歇性運動型式,2分鐘階段間休息1分鐘。第一次運動後進行1小時的恢復期,在第二次運動後進行2小時的恢復期。而在第二次運動後立即補充1g/kg碳水化合物+0.1 g/kg 精胺酸+0.1 g/kg 支鏈胺基酸(GLU+AA trial)、1.2 g/kg 碳水化合物(GLU trial)、或安慰劑(PLA trial)。血液採集和氣體分析於早餐食用前、第一次運動前、第一次運動後0分鐘、30分鐘、60分鐘、第二個階段運動後0、30分、60分、90分、120分鐘、第三個運動階段後。氣體樣本分析項目為碳水化合物氧化率、脂肪氧化率;血液樣本分析項目為血漿中葡萄糖、胰島素、非酯化脂肪酸、甘油、乳酸、氨、肌酸激酶、乳酸脫氫酶。結果顯示三個trial的運動表現並沒有顯著的差異,各trial在各次運動總平均功率無顯著差異(EX1:GLU+AA trial 64.24 ±4.14 W/kg;GLU trial 63.90 ±3.82W/kg;PLA trial 61.97 ±3.33W/kg,EX2:GLU+AA trial 63.48 ±5.54 W/kg;GLU trial 61.05 ±4.59 W/kg;PLA trial 61.41 ±4.84 W/kg;EX3:GLU+AA trial63.85 ±7.09 W/kg;GLU trial 60.89 ±4.42 W/kg;PLA trial 59.27 ±4.15 W/kg),三階段總最大功率亦無顯著差異。補充飲料後第60、90分鐘GLU+AA trail碳水化合物氧化率顯著高於PLA trial,GLU trial第60分鐘顯著高於PLA組。各運動階段後碳水化合物曲線下面積,3個trial間無顯著差異。補充飲料後第60、90分鐘GLU+AA trial脂肪氧化率顯著低於PLA trial,而GLU trial第90、120分鐘顯著低於PLA trial,各運動階段後脂肪氧化率曲線下面積,GLU+AA trial與GLU trial顯著低於PLA組。補充後第30分鐘GLU+AA trail與GLU trial血漿中葡萄糖濃度顯著大於PLA trial,第二運動階段後恢復期血漿中葡萄糖曲線下面積,GLU+AA trial與GLU trial顯著高於PLA組。補充飲料後第30、60、90分鐘GLU+AA trial 胰島素濃度顯著高於PLA trial,GLU trial第30分鐘顯著高於PLA trial。第二運動階段後恢復期血漿中胰島素曲線下面積,GLU+AA trial與GLU trial顯著高於PLA組。非酯化脂肪酸與甘油濃度為補充飲料後第90、120分鐘及第三階段運動後GLU+AA trial顯著低於PLA trial,而GLU trial亦顯著低於PLA trial。乳酸、氨、肌酸激酶與乳酸脫氫酶濃度在各trial之間則無顯著差異。研究結果顯示高強度間歇運動後補充碳水化合物可以提高血糖與胰島素濃度、碳水化合物氧化率,並降低血漿中NEFA、Glycerol濃度、脂肪氧化率,但補充支鏈胺基酸與精胺酸無加成效果,且對於後續運動表現並無顯著的影響。 | |
| dc.description.abstract | Branched-chain amino acids (BCAA) and arginine (ARG) have a wide range of physiological functions that may improve exercise performance. BCAA may stimulate the insulin response to increase muscle glycogen recovery and reduce skeletal muscle protein proteolysis. Arginine may stimulate endothelium-dependent vasodilation and reduce exercise-induced blood lactate and ammonia accumulation. The purpose of this study was to investigate the effect of BCAA and ARG supplementation on recovery after intermittent high-intensity exercise and performance in the subsequent exercise. The potential biochemical mechanisms were also explored. Nine male college wrestlers were recruited. All subjects completed 3 experimental trials in a random order. The intermittent anaerobic test was consisted of 3 rounds with 4 sets in each round. The subjects alternated 10-sec all-out exercise and 20-sec periods on a cycle ergometer. There was 1 min rest between each round. The load in the exercise period was set 0.1 kp/kg. There was a 1-hr recovery period after the first exercise test, and 2-hr recovery period after the second exercise test. After the second exercise test, the subjects consumed 1 g/kg glucose plus 0.1 g/kg arginine and 0.1 g/kg BCAA (leucine:isoleucine:valine=2:1:1)(GLU+AA trial), 1.2 g/kg glucose (GLU trial), or Placebo (PLA trial). The blood and expired gas samples were analyzed before breakfast, immediately before and after the first exercise, 30 and 60 min after the first exercise, 0, 30, 60, 90 and 120 min after the second exercise, and immediately after the third exercise. Carbohydrate and fat oxidation rates were calculated from the results of gas analysis. The plasma sample were used to measure glucose, insulin, nonesterified fatty acids (NEFA), glycerol, lactate, NH3, creatine kinase (CK), lactate dehydrogenase (LDH). The results showed that there was no difference in exercise performance among the 3 trials. Total average power was similar among the 3 trials (EX1: GLU + AA trial 64.24 ± 4.14 W / kg; GLU trial 63.90 ± 3.82W/kg; PLA trial 61.97 ± 3.33W/kg, EX2: GLU + AA trial 63.48 ± 5.54 W / kg; GLU trial 61.05 ± 4.59 W / kg; PLA trial 61.41 ± 4.84 W / kg; EX3: GLU + AA trial63.85 ± 7.09 W / kg; GLU trial 60.89 ± 4.42 W / kg; PLA trial 59.27 ± 4.15 W / kg). Total peak power was also similar among the 3 trials. GLU + AA trail had significantly higher carbohydrate oxidation rate at 60 and 90 min postprandial than PLA trial. GLU trial had significantly higher carbohydrate oxidation rate at 60 min postprandial than PLA trial. The area under the curve (AUC) of carbohydrate oxidation rate was similar among the 3 trials. GLU + AA trail had significantly lower fat oxidation rate at 60 and 90 min postprandial than PLA trial. GLU trial had significantly lower fat oxidation at 60 and 120 postprandial min than PLA trial. The AUC of fat oxidation rate was significantly lower in GLU + AA and GLU trial than PLA trial. GLU + AA and GLU trial had significantly higher plasma glucose concentration at 30 min postprandial than PLA trial. The plasma glucose AUC after the second exercise was significantly higher in GLU + AA and GLU trial than that in PLA trial. GLU + AA trail had significantly higher plasma insulin concentration at 30, 60, and 90 min postprandial than PLA trial. GLU trial had significantly higher plasma insulin concentration at 30 min postprandial than PLA trial. The plasma insulin AUC after the second exercise was significantly higher in GLU + AA trail and GLU trial than that in PLA trial. GLU + AA and GLU trial had significantly lower plasma NEFA and glycerol concentrations at 90 and 120 min after the second exercise and immediately after the third exercise than PLA trial. There were no differences in plasma lactate concentration, NH3 concentration, CK concentration, and LDH concentration among the 3 trials. The current results suggested that the supplementation of carbohydrate can increase plasma glucose and insulin concentration and carbohydrate oxidation rate,while reducing plasma NEFA and Glycerol concentrations and fat oxidation. However, BCAA and arginine supplementation showed no additional effect on substrate metabolism and the performance in the subsequent exercise after intermittent high-intensity exercise. | |
| dc.description.tableofcontents | 目 次 第一章 緒論……………………………………………………………1 第一節 研究背景……………………………………………………1 第二節 研究目的……………………………………………………2 第三節 研究假設……………………………………………………3 第二章 文獻探討………………………………………………………4 第一節 運動對肌肉肝醣的影響……………………………………4 第二節 支鏈胺基酸對運動表現的影響……………………………7 第三節 精胺酸與運動表現的影響…………………………………12 第三章 研究方法與步驟………………………………………………15 第一節 研究對象……………………………………………………15 第二節 實驗設計……………………………………………………15 第三節 運動測試……………………………………………………16 第四節 實驗方法……………………………………………………17 第五節 資料處理與統計分析………………………………………20 第四章 結果……………………………………………………………21 第五章 討論……………………………………………………………25 第六章 結論與建議……………………………………………………32 第一節 結論…………………………………………………………32 第二節 建議…………………………………………………………33 參考文獻…………………………………………………………………34 | |
| dc.format.extent | 438158 bytes | |
| dc.format.mimetype | application/pdf | |
| dc.identifier.uri | https://ir.ntus.edu.tw/handle/987654321/70986 | |
| dc.language | zh-TW | |
| dc.publisher | 競技運動學系碩士班 | |
| dc.relation.isbasedon | Asmussen, E. (1993). Muscle fatigue. Medicine and Science in Sports and Exercis, 25, 411-20,. Balsom PD, Gaitanos GC, Solund K, Ekblom B. (1999). High-intensity exercise and muscle glycogen availability in humans. Acta Physiologica Scandinavica, 165(4), 337-345. Bangsbo, J., Madsen, K., Kiens, B. & Richter, E. A. (1996). Effect of muscle acidity on muscle metabolism and fatigue during intense exercise in man. The Journal of Physiology. 495, 587-596. Bangsbo, J., Nùrregaard, L. & Thorsùe, F. (1992). The effect of carbohydrate diet on intermittent exercise performance. International Journal of Sports Medicine, 13, 152-157. Bednarz, B., Wolk, R. & Chamiec, T. (2000). Effects of oral L-arginine supplementation on exercise-induced QT dispersion and exercise tolerance in stable angina pectoris. International Journal of Cardiology, 75, 205-210. Bergstrom, J., Hermansen, L., Hultman, E. & Saltin, B. (1967). Diet, muscle glycogen and physical performance. Acta Physiologica Scandinavica, 71(2), 140-150. Betts, J. A., Stevenson E, Williams C, et al. (2005). Recovery of endurance running capacity: effect of carbohydrate-protein mixtures. International Journal of Sport Nutrition and Exercise Metabolism 15: 590-609. Betts, J., Williams, C., Duffy, K., Gunner, F. (2007). The influence of carbohydrate and protein ingestion during recovery from prolonged exercise on subsequent endurance performance. Journal of Sports Sciences, 25, 1449-1460. Blomstrand E, Celsing F, Newsholme EA. (1988). Changes in plasma concentrations of aromatic and branched-chain amino acids during sustained exercise in man and their possible role in fatigue. Acta Physiologica Scandinavica, 133, 115-21. Blomstrand E, Hassmen P, Ek S, Ekblom B, Newsholme EA. (1997). Influence of ingesting a solution of branched-chain amino acids on perceived exertion during exercise. Acta Physiologica Scandinavica,159, 41-9. Ceremuzynski L, Chamiec T, Herbaczynska-Cedro K. (1997). Effect of supplemental oral L-arginine on exercise capacity in patients with stable angina pectoris. The American Journal of Cardiology, 80, 331-3. Cheng JW, Baldwin SN, Balwin SN. (2001). L-arginine in the management of cardiovascular diseases. The Annals of Pharmacotherapy, 35, 755-64. Coombes JS, McNaughton LR. (2000). Effects of branched-chain amino acid supplementation on serum creatine kinase and lactate dehydrogenase after prolonged exercise. The Journal of Sports Medicine and Physical Fitness, 40, 240-6. Corson MA, James NL, Latta SE, et al. (1996). Phosphorylation of endothelial nitric oxide synthase in response to fluid shear stress. Circulation Research, 79, 984-91. Curzon G, Friedel J, Knott PJ. (1973). The effect of fatty acids on the binding of tryptophan to plasma protein. Nature, 242, 198-200. Davis JM, Bailey SP. (1997). Possible mechanisms of central nervous system fatigue during exercise. Medicine and Science in Sports and Exercise, 29, 45-57. Davis JM, Welsh RS, De Volve KL, Alderson NA. (1999). Effects of branched-chain amino acids and carbohydrate on fatigue during intermittent, high-intensity running. International Journal of Sports Medicine, 20, 309-14. Denis C, Dormois D, Linossier MT, et al. (1991). Effect of arginine aspartate on the exercise-induced hyperammoniemia in humans: a two periods cross-over trial. Archives Internationales De Physiologie, De Biochimie Et De Biophysique, 99, 123-7. Eto B, Peres G, Le Moel G. (1994). Effects of an ingested glutamate arginine salt on ammonemia during and after long lasting cycling. Archives Internationales De Physiologie, De Biochimie Et De Biophysique, 102, 161-2. Fallowfield JL, Williams C, Singh R. (1995). The influence of ingesting a carbohydrate-electrolyte beverage during 4 hours of recovery on subsequent endurance capacity. International Journal of Sport Nutrition, 5, 285-99. Favero TG, Zable AC, Colter D, Abramson JJ. (1997). Lactate inhibits Ca(2+) -activated Ca(2+)-channel activity from skeletal muscle sarcoplasmic reticulum. Journal of Applied Physiology, 82, 447-52. Fernstrom JD, Faller DV. (1978). Neutral amino acids in the brain: changes in response to food ingestion. Journal of Neurochemistry, 30, 1531-8. Fernstrom JD, Wurtman RJ. (1972). Brain serotonin content: physiological regulation by plasma neutral amino acids. Science (New York, N.Y.), 178, 414-6. Fernstrom JD. (2005). Branched-chain amino acids and brain function. The Journal of Nutrition, 135, 1539S-46S. Fujita H, Yamabe H, Yokoyama M. (2000). Effect of L-arginine administration on myocardial thallium-201 perfusion during exercise in patients with angina pectoris and normal coronary angiograms. Journal of nuclear cardiology : official publication of the American Society of Nuclear Cardiology, 7, 97-102. Gaitanos, G.C., Williams, C., Boobis, L.H. & Brooks, S. (1993). Human muscle metabolism during intermittent maximal exercise. Journal of Applied Physiology, 75, 712±719. Gandevia SC. (2001). Spinal and supraspinal factors in human muscle fatigue. Physiological Reviews, 81, 1725-89. Gielen S, Schuler G, Hambrecht R. (2001). Exercise training in coronary artery disease and coronary vasomotion. Circulation, 103, E1-6. Gomez-Merino D, Bequet F, Berthelot M, et al. (2001). Evidence that the branched-chain amino acid L-valine prevents exercise-induced release of 5-HT in rat hippocampus. International Journal of Sports Medicine, 22, 317-22. Goto C, Nishioka K, Umemura T, et al. (2007). Acute moderate-intensity exercise induces vasodilation through an increase in nitric oxide bioavailiability in humans. American Journal of Hypertension, 20, 825-30. Harper AE, Miller RH, Block KP. (1984). Branched-chain amino acid metabolism. Annual Review of Nutrition, 4, 409-54. Hassmen P, Blomstrand E, Ekblom B, Newsholme EA. (1994). Branched-chain amino acid supplementation during 30-km competitive run: mood and cognitive performance. Nutrition, 10, 405-10. Hellsten Y. (1999). The effect of muscle contraction on the regulation of adenosine formation in rat skeletal muscle cells. The Journal of physiology, 518, 761-8. Hockachka, P. W. & G. N. Somero. (1984). Biochemical Adaptation, Princeton, Nutrition Journal: Princeton University Press. Ivy, J. L. (1999). Role of carbohydrate in physical activity. Clinics in sports medicine, 18(3), 469-484. Ivy, J. L. (2004). Timing and optimization of dietary supplements for recovery and performance. Journal of Exercise Science and Fitness, 2(2), 79-84. Ivy, J. L., Katz, A. L., Cutler, C. L., Sherman, W. M. & Coyle, E. F. (1988). Muscle glycogen synthesis after exercise: Effect of time of carbohydrate ingestion. Journal of Applied Physiology, 64(4), 1480-1485. Katz A, Sahlin K, Henriksson J. (1986). Muscle ammonia metabolism during isometric contraction in humans. The American Journal of Physiology, 250, C834-40. Keizer HA, Kuiperes H, van kranenburg G, etal. (1986). Influence of liquid and solid meals on muscle glycogen resynthesis, plasma fuel hormone response, and maximal physical working capcity. International Journal of Sports Medicine, 8(2):99-104. Kingwell BA. (2000). Nitric oxide as a metabolic regulator during exercise: effects of training in health and disease. Clinical and Experimental Pharmacology &Pphysiology, 27, 239-50. Laughlin MH. (1995). Endothelium-mediated control of coronary vascular tone after chronic exercise training. Medicine and Science in Sports and Exercise, 27, 1135-44. Levenhagen, D. K., Gresham, J. D., Carlson, M. G., Maron, D. J., Borel, M. J., & Flakoll, P. J. (2001). Postexercise nutrient intake timing in humans is critical to recovery of leg glucose and protein homeostasis. American Journal of Physiology. Endocrinology and metabolism, 280(6), E982-993. Lira VA, Soltow QA, Long JH, et al. (2007). Nitric oxide increases GLUT4 expression and regulates AMPK signaling in skeletal muscle. American Journal of Physiology Endocrinology and Metabolism 293, E1062-8. Lund S, Holman GD, Schmitz O and Pedersen O (1993) Glut 4 content in the plasma membrane of rat skeletal muscle: Comparative studies of the subcellular fraction-method and the exofacial photolabelling technique using ATB-BMPA. FEBS, 330, 312-318. MacLean DA, Graham TE, Saltin B. (1994). Branched-chain amino acids augment ammonia metabolism while attenuating protein breakdown during exercise. The American Journal of Physiology, 267, E1010-22. Madsen K, MacLean DA, Kiens B, Christensen D. (1996). Effects of glucose, glucose plus branched-chain amino acids, or placebo on bike performance over 100 km. Journal of Applied Physiology, 81, 2644-50. Maehlum, S., Hostmark, A. T. and Hermansen. L. (1977). Synthesis of muscle glycogen during recovery after prolonged severe exercise in diabetic and non-diabetic subjects. Scandinavian Journal of Clinical & Laboratory Investigation. 37, 309-316. Matsumoto K, Mizuno M, Mizuno T, et al. (2007). Branched-chain amino acids and arginine supplementation attenuates skeletal muscle proteolysis induced by moderate exercise in young individuals. International Journal of Sports Medicine, 28, 531-8. Meeusen R, Watson P, Dvorak J. (2006). The brain and fatigue: new opportunities for nutritional interventions? Journal of sports sciences, 24, 773-82. Millard-Stafford M, Warren GL, Thomas LM, et al. (2005). Recovery from run training: efficacy of a carbohydrate-protein beverage? International Journal of Sport Nutrition and Exercise Metabolism, 15, 610-24. Mittleman KD, Ricci MR, Bailey SP. (1998). Branched-chain amino acids prolong exercise during heat stress in men and women. Medicine and Science in Sports and Exercise, 30, 83-91. Moncada S, Higgs A. (1993). The L-arginine-nitric oxide pathway. The New England Journal of Medicine, 329, 2002-12. Muller JM, Myers PR, Laughlin MH. (1994). Vasodilator responses of coronary resistance arteries of exercise-trained pigs. Circulation, 89, 2308-14. Mutch BJ, Banister EW. (1983). Ammonia metabolism in exercise and fatigue: a review. Medicine and Science in Sports and Exercise, 15, 41-50. Newsholme EA, Blomstrand E. (2006). Branched-chain amino acids and central fatigue. The Journal of Nutrition, 136, 274S-6S. Nygaard K. (1978). Trade union movement and ADP development: ADP will result in new and extended authority for the employer. Sygeplejersken, 29;78(47):suppl 4-8 Pardridge WM. (1998). Blood-brain barrier carrier-mediated transport and brain metabolism of amino acids. Neurochemical Research, 23, 635-44. Pardridge WM. (1979). The role of blood-brain barrier transport of tryptophan and other neutral amino acids in the regulation of substrate-limited pathways of brain amino acid metabolism. Journal of Neural Transmission Supplementum, 43-54. Pernow B, Saltin B. (1971) Availability of substrates and capacity for prolonged heavy exercise in man. Journal of Applied Physiology.Sep;31(3):416-22. Price, T. B., Rothman, D. L., Taylor, R., Avison, M. J., Shulman, G. I. and Shulman, R. G. (1994). Human muscle glycogen resynthesis after exercise: insulin-dependent and -independent phases. Journal of Applied Physiology, 76, 104-11. Rector TS, Bank AJ, Mullen KA, et al. (1996). Randomized, double-blind, placebo-controlled study of supplemental oral L-arginine in patients with heart failure. Circulation, 93, 2135-41. Ren, J. M., Marshall, B. A., Gulve, E. A., GaoJ, Johnson, D. W. & Holloszy, J. O. (1993). Evidence from transgenic mice that glucose transport is rate-limiting for glycogen deposition and glycolysis in skeletal muscle. The Journal of Biological Chemistry, 268, 16113-16115. Riazi R, Wykes LJ, Ball RO, Pencharz PB. (2003). The total branched-chain amino acid requirement in young healthy adult men determined by indicator amino acid oxidation by use of L-[1-13C]phenylalanine. Journal of Nutrition, 133, 1383-9. Romijn, J. A., Coyle, E. F., Sidossis, L. S., Gastaldelli, A., Horowitz, J. F., Endert, E. et al. (1993). Regulation of endogenous fat and carbohydrate metabolism in relation to exercise intensity and duration. American Journal of Physiology. Endocrinology and Metabolism, 265(3), E380-391. Schaefer A, Piquard F, Geny B, et al. (2002). L-Arginine reduces exercise-induced increase in plasma lactate and ammonia. International Journal of Sports Medicine, 23, 403-407. Schrage WG, Eisenach JH, Joyner MJ. (2007). Ageing reduces nitric-oxide- and prostaglandin-mediated vasodilatation in exercising humans. The Journal of Physiology, 579, 227-36. Sessa WC, Pritchard K, Seyedi N, Wang J, Hintze TH. (1994). Chronic exercise in dogs increases coronary vascular nitric oxide production and endothelial cell nitric oxide synthase gene expression. Circulation Research 74, 349-53. Shimomura Y, Fujii H, Suzuki M, et al. (1995). Branched-chain alpha-keto acid dehydrogenase complex in rat skeletal muscle:regulation of the activity and gene expression by nutrition and physical exercise. Journal of Nutrition 125, 1762S-1765S. Shimomura Y, Murakami T, Nakai N, Nagasaki M, Harris RA. (2004). Exercise promotes BCAA catabolism: effects of BCAA supplementation on skeletal muscle during exercise. Journal of Nutrition 134, 1583S-1587S. Spodaryk K, Szmatlan U, Berger L. (1990). The relationship of plasma ammonia and lactate concentrations to perceived exertion in trained and untrained women. European Journal of Applied Physiology and Occupational Physiology 61, 309-12. Stevens BR, Godfrey MD, Kaminski TW, Braith RW. (2000). High-intensity dynamic human muscle performance enhanced by a metabolic intervention. Medicine and Science in Sports and Exercise 32, 2102-8. Struder HK, Hollmann W, Platen P, et al. (1998). Influence of paroxetine, branched-chain amino acids and tyrosine on neuroendocrine system responses and fatigue in humans. Hormone and Metabolic Research 30, 188-94. Talanian J L, Galloway S D, Heigenhauser G J, Bonen A, & Spriet L L. (2007). Two weeks of high-intensity aerobic interval training increases the capacity for fat oxidation during exercise in women. Journal of Applied Physiology, 102(4), 1439-1447. van Loon LJ, Kruijshoop M, Verhagen H, Saris WH, Wagenmakers AJ. (2000). Ingestion of protein hydrolysate and amino acid-carbohydrate mixtures increases postexercise plasma insulin responses in men. Journal of Nutrition 130, 2508-13. van Loon LJ, Saris WH, Kruijshoop M, Wagenmakers AJ. (2000). Maximizing postexercise muscle glycogen synthesis: carbohydrate supplementation and the application of amino acid or protein hydrolysate mixtures. The American Journal of Clinical Nutrition 72, 106-11. van Loon LJ, Saris WH, Verhagen H, Wagenmakers AJ. (2000). Plasma insulin responses after ingestion of different amino acid or protein mixtures with carbohydrate. The American Journal of Clinical Nutrition 72, 96-105. Wagenmakers AJ, Brookes JH, Coakley JH, Reilly T, Edwards RH. (1989). Exercise-induced activation of the branched-chain 2-oxo acid dehydrogenase in human muscle. European Journal of Applied Physiology and Occupational Physiology 59, 159-67. Wang J, Wolin MS, Hintze TH. (1993). Chronic exercise enhances endothelium-mediated dilation of epicardial coronary artery in conscious dogs. Circulation Research 73, 829-38. Williams MB, Raven PB, Fogt DL, Ivy JL. (2003). Effects of recovery beverages on glycogen restoration and endurance exercise performance. Journal of Strength and Conditioning Research / National Strength & Conditioning Association 17, 12-9. | |
| dc.subject | 支鏈胺基酸;精胺酸;肝醣;高強度間歇運動;運動表現 | |
| dc.subject | branched-chain amino acids;arginine;glycogen;high-intensity exercise;exercise performance | |
| dc.title | 補充支鏈胺基酸與精胺酸對高強度間歇運動後恢復與後續運動表現的影響 | |
| dc.title | THE EFFECT OF BRANCHED-CHAIN AMINO ACIDS AND ARGININE SUPPLEMENTATION ON RECOVERY AFTER INTERMITTENT HIGH-INTENSITY EXERCISE AND PERFORMANCE IN THE SUBSEQUENT EXERCISE | |
| dc.type | thesis | |
| dspace.entity.type | Publication |
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